Type 1 diabetes is an autoimmune disorder in which the body's immune system destroys the insulin-producing beta cells of the pancreas, leading to little or no production of insulin, the hormone required to move glucose from the blood into cells for energy. Without sufficient insulin, glucose accumulates in the blood…
Type 1 diabetes is an autoimmune disorder in which the body's immune system destroys the insulin-producing beta cells of the pancreas, leading to little or no production of insulin, the hormone required to move glucose from the blood into cells for energy. Without sufficient insulin, glucose accumulates in the bloodstream, and the condition, if unmanaged, can cause acute complications such as diabetic ketoacidosis and, over time, damage to the eyes, kidneys, nerves, and cardiovascular system. Type 1 diabetes typically develops in childhood or young adulthood, has no cure, and requires lifelong management with insulin replacement, blood-glucose monitoring, and attention to diet and lifestyle. As a hormonal and metabolic disease, it is a central subject of endocrinology research. Related peer-reviewed work in this collection includes a study suggesting that residual beta-cell function in type 1 diabetes may be greater than presumed, an investigation of reduced tissue oxygenation and altered cardiovascular responses in young adults with type 1 diabetes, and an examination of the impact of nutrients on diabetes. This page gathers open-access research relevant to the autoimmune destruction, physiology, complications, and management of type 1 diabetes within the broader field of Endocrinology And Hormones.
12 peer-reviewed articles, ranked by relevance. Each links to its DOI.
The 12 articles above have been cited 43 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.
A sample of recent works citing this journal's research on Type 1 Diabetes, linking to each citing work.