Overview
Apoptosis is a regulated, energy-dependent form of programmed cell death by which multicellular organisms eliminate unwanted, damaged, or potentially harmful cells in a controlled manner. It is essential for normal development, tissue homeostasis, and immune regulation, and it serves as a defense against cells bearing dangerous mutations. Morphologically, apoptosis is characterized by cell shrinkage, chromatin condensation, nuclear fragmentation, and the formation of membrane-bound apoptotic bodies that are cleared without provoking inflammation, distinguishing it from necrosis. The process is executed through tightly coordinated signaling pathways, including an intrinsic mitochondrial pathway responsive to cellular stress and an extrinsic pathway triggered by death-receptor activation, both converging on proteolytic enzymes that dismantle the cell. Dysregulation of apoptosis has broad pathological significance: insufficient cell death contributes to tumor formation and resistance to therapy, whereas excessive death is implicated in degenerative and ischemic injury. Research in this area examines how chemical agents, oxidative stress, and metabolic conditions modulate cell survival and death, how apoptotic regulation can be exploited in cancer prevention and treatment, and how proteins governing the death program influence processes such as metastasis. Antioxidant and protective compounds are also studied for their capacity to limit damage-induced cell death. Understanding apoptosis is therefore central to developmental biology, oncology, and the design of therapies that restore appropriate control of cell survival.
Research published in this journal
12 peer-reviewed articles, ranked by relevance. Each links to its DOI.
NNC55-0396 Reduces SH-SY5Y Cell Damage Induced by Bupivacaine Hydrochloride
The Novel Coronavirus 2019 (COVID-19): A Narrative Review
Insights on the Thermal and Physical Stability of the Modified Polymerizable Liposomes for Improved Photoactivity
Radical-Scavenging and Anti-Oxidative Activities of TBN in Cell-Free System and Murine H9c2 Cardiomyoblast Cells
A Cancer Theory: The Central Nervous System’s Adaptive Changes Make Chronic Diseases Incurable
Correlating 13C Isotope in Oligomeric Proanthocyanidins with their Anticancer Properties
First Evidences of Epithelial-Mesenchymal Transition and Cancer Stem-Cell Phenotype Acquisition in Dermo-Epidermal Junction of BPV-Infected Neoplasms
MicroRNA: New Era for Therapeutic Strategy in Ischaemic Heart Disease
Ozone/Oxygen Molecules Exert Mild Oxidative Stress on Testis Mitochondria Isolated from the Rat Testicular Ischemia/Reperfusion Injury
Overexpression of Prostate Apoptosis Response Protein-4 In Colon Cancer Cells Can Inhibit Metastasis by Upregulating E-cadherin Expression
How this research is being cited
The 12 articles above have been cited 41 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.
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2026 · AAPS PharmSciTech
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2025 · Journal of Drug Delivery Science and Technology
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2025 · Journal of Drug Delivery Science and Technology
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2024 · Journal of Biomedical Materials Research Part A
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Vitor Rodrigues da Costa et al. · 2023 · Exploration of Immunology
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2023 · Exploration of Immunology
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Jaekwon Seok et al. · 2023 · Journal of Translational Medicine
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2023 · Journal of Biomedical Materials Research Part A
A sample of recent works citing this journal's research on Cell Apoptosis, linking to each citing work.